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This can be a 3-in-1 reference booklet. It offers an entire scientific dictionary protecting thousands of phrases and expressions on the subject of chloramphenicol. It additionally supplies huge lists of bibliographic citations. ultimately, it offers info to clients on how one can replace their wisdom utilizing a number of net assets. The publication is designed for physicians, scientific scholars getting ready for Board examinations, clinical researchers, and sufferers who are looking to get to grips with examine devoted to chloramphenicol. in the event that your time is effective, this e-book is for you. First, you won't waste time looking the web whereas lacking loads of appropriate details. moment, the e-book additionally saves you time indexing and defining entries. ultimately, you won't waste money and time printing hundreds of thousands of websites.
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Extra info for Chloramphenicol - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References
Author(s): Polak BC, Wesseling H, Schut D, Herxheimer A, Meyler L. Source: Acta Med Scand. 1972 November; 192(5): 409-14. No Abstract Available. cmd=Retrieve&db=pubmed&dopt=A bstract&list_uids=5083381 • Blood dyscrasias attributed to chloramphenicol: a review of 641 published and unpublished cases. Author(s): Polak BC, Wesseling H, Schut D, Herxheimer A. Source: Postgraduate Medical Journal. 1974 October; 50 Suppl 5: 123-6. cmd=Retrieve&db=pubmed&dopt=A bstract&list_uids=4470805 • Blood level study of chloramphenicol and its formulation with amphotericin-B andor ascorbic acid.
Gram-negative bacterial biofilm formation is dependent upon the synthesis of quorum-sensing transcriptional activators, called autoinducers. We have found that H. influenzae; including those isolated from the middle ear possess a gene (HI0491) capable of synthesizing an autoinducer (AI-2). Insertional inactivation of HI0491 in the H. influenzae laboratory strain Rd KW20 results in a mutant, which lacks the ability to form mature biofilm structures, and has decreased susceptibility to antibiotics, a decreased conjugation frequency and decreased survival in an animal model of OME.
The transformation from a diploid trophoblast to a giant cell is hypothesized to be induced by factors that also regulate trophoblast giant cell-specific gene expression. Two such transcription factors are GATA-2 and GATA-3. Experiments are proposed to test the possibility that GATA-2 and GATA-3 are determinants in the switch from proliferative trophoblasts to terminally differentiated giant cells in culture, and regulators of placental development in vivo. Trophoblast cells will be manipulated in culture and in vivo to increase or decrease GATA factor activity, and the consequence of these changes on cell differentiation and development will be examined.